Presenilin 1 deficiency suppresses autophagy in human neural stem cells through reducing γ-secretase-independent ERK/CREB signaling-Reference-Cited by-同舟云学术

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Presenilin 1 deficiency suppresses autophagy in human neural stem cells through reducing γ-secretase-independent ERK/CREB signaling

Published:2018-08-29 Issue:9 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Chong Cheong-Meng,Ke Minjing,Tan Yuan,Huang Zhijian,Zhang Ke,Ai Nana,Ge Wei,Qin Dajiang,Lu Jia-Hong,Su Huanxing

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-0945-7.pdf

Reference50 articles.

1. Alzheimer’s, A. 2016 Alzheimer’s disease facts and figures. Alzheimers Dement. 12, 459–509 (2016).

2. Janssen, J. C. et al. Early onset familial Alzheimer’s disease: mutation frequency in 31 families. Neurology 60, 235–239 (2003).

3. Kovacs, D. M. et al. Alzheimer-associated presenilins 1 and 2: neuronal expression in brain and localization to intracellular membranes in mammalian cells. Nat. Med. 2, 224–229 (1996).

4. Suzuki, T. et al. Regional and cellular presenilin 1 gene expression in human and rat tissues. Biochem. Biophys. Res. Commun. 219, 708–713 (1996).

5. Lee, M. K. et al. Expression of presenilin 1 and 2 (PS1 and PS2) in human and murine tissues. J. Neurosci. 16, 7513–7525 (1996).

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