Galunisertib suppresses the staminal phenotype in hepatocellular carcinoma by modulating CD44 expression
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology
Link
http://www.nature.com/articles/s41419-018-0384-5.pdf
Reference34 articles.
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2. Snowberger, N. et al. Alpha fetoprotein, ultrasound, computerized tomography and magnetic resonance imaging for detection of hepatocellular carcinoma in patients with advanced cirrhosis. Aliment. Pharmacol. Ther. 26, 1187–1194 (2007).
3. Severi, T., Malenstein, H. V., Verslype, C. & Pelt, J. V. Tumor initiation and progression in hepatocellular carcinoma: risk factors, classification, and therapeutic targets. Acta Pharmacol. Sin. 31, 1409–1420 (2010).
4. Giannelli, G. et al. The rationale for targeting TGF-β in liver diseases. Eur. J. Clin. Invest. 46, 349–361 (2016).
5. Cao, Y. et al. NGS-based transcriptome profiling reveals biomarkers for companion diagnostics of the TGF-β receptor blocker galunisertib in HCC. Cell Death Dis. 8, e2634 (2017).
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