Fas-L promotes the stem cell potency of adipose-derived mesenchymal cells-Reference-Cited by-同舟云学术

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Fas-L promotes the stem cell potency of adipose-derived mesenchymal cells

Published:2018-06 Issue:6 Volume:9 Page:
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Solodeev Inna,Meilik Benjamin,Volovitz Ilan,Sela Meirav,Manheim Sharon,Yarkoni Shai,Zipori Dov,Gur Eyal,Shani Nir

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/s41419-018-0702-y.pdf

Reference52 articles.

1. Strasser, A., Jost, P. J. & Nagata, S. The many roles of FAS receptor signaling in the immune system. Immunity 30, 180–192 (2009).

2. Kischkel, F. et al. Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor. EMBO J. 14, 5579 (1995).

3. Boldin, M. P., Goncharov, T. M., Goltseve, Y. V. & Wallach, D. Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1-and TNF receptor–induced cell death. Cell 85, 803–815 (1996).

4. Medema, J. P. et al. FLICE is activated by association with the CD95 death‐inducing signaling complex (DISC). EMBO J. 16, 2794–2804 (1997).

5. Muzio, M. et al. FLICE, a novel FADD-homologous ICE/CED-3–like protease, is recruited to the CD95 (Fas/APO-1) death-inducing signaling complex. Cell 85, 817–827 (1996).

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