IL-38 has an anti-inflammatory action in psoriasis and its expression correlates with disease severity and therapeutic response to anti-IL-17A treatment
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology
Link
http://www.nature.com/articles/s41419-018-1143-3.pdf
Reference49 articles.
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2. Zheng, Y. et al. Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis. Nature 445, 648–651 (2007).
3. Ogawa, E., Sato, Y., Minagawa, A. & Okuyama, R. Pathogenesis of psoriasis and development of treatment. J. Dermatol. 45, 264–272 (2018).
4. Madonna, S., Scarponi, C., Pallotta, S., Cavani, A. & Albanesi., C. Anti-apoptotic effects of suppressor of cytokine signaling 3 and 1 in psoriasis. Cell Death Dis. 3, e334 (2012).
5. van de Veerdonk, F. L., de Graaf, D. M., Joosten, L. A. & Dinarello, C. A. Biology of IL-38 and its role in disease. Immunol. Rev. 281, 191–196 (2018).
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