A putative nitroreductase from the DosR regulon of Mycobacterium tuberculosis induces pro-inflammatory cytokine expression via TLR2 signaling pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep24535.pdf
Reference54 articles.
1. Forrellad, M. A. et al. Virulence factors of the Mycobacterium tuberculosis complex. Virulence 4, 3–66 (2013).
2. Guirado, E. et al. Characterization of host and microbial determinants in individuals with latent tuberculosis infection using a human granuloma model. MBio 6, e02537–02514 (2015).
3. Watanabe, S. et al. Fumarate reductase activity maintains an energized membrane in anaerobic Mycobacterium tuberculosis. PLoS Pathog 7, e1002287 (2011).
4. Leyten, E. M. et al. Human T-cell responses to 25 novel antigens encoded by genes of the dormancy regulon of Mycobacterium tuberculosis. Microbes Infect 8, 2052–2060 (2006).
5. Sasindran, S. J. & Torrelles, J. B. Mycobacterium Tuberculosis Infection and Inflammation: what is Beneficial for the Host and for the Bacterium? Front Microbiol 2, 2 (2011).
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