Type 1 iodothyronine deiodinase is the major source of circulating T3 in hyperthyroidism: implications for therapy
Author:
Publisher
Springer Science and Business Media LLC
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
http://www.nature.com/articles/ncpendmet0619.pdf
Reference5 articles.
1. Bianco AC et al. (2002) Biochemistry, cellular and molecular biology, and physiological roles of the iodothyronine selenodeiodinases. Endocr Rev 23: 38–89
2. Maia AL et al. (2005) Type 2 iodothyronine deiodinase is the major source of plasma T3 in euthyroid humans. J Clin Invest 115: 2524–2533
3. Abuid J and Larsen PR (1974) Triiodothyronine and thyroxine in hyperthyroidism. Comparison of the acute changes during therapy with antithyroid agents. J Clin Invest 54: 201–208
4. Cooper DS (2005) Antithyroid drugs. N Engl J Med 352: 905–917
5. Nakamura H (2007) Comparison of methimazole and propylthiouracil in patients with hyperthyroidism caused by Graves' disease. J Clin Endocrinol Metab 92: 2157–2162
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Type 1 iodothyronine deiodinase in human physiology and disease;Journal of Endocrinology;2011-03-17
2. Central and Peripheral Effects of Thyroid Hormone Signalling in the Control of Energy Metabolism;Journal of Neuroendocrinology;2010-01
3. Impacto do tratamento com GH sobre as funções tireoidiana e adrenal;Arquivos Brasileiros de Endocrinologia & Metabologia;2008-07
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