RAF proteins exert both specific and compensatory functions during tumour progression of NRAS-driven melanoma
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry
Link
http://www.nature.com/articles/ncomms15262.pdf
Reference51 articles.
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2. Fedorenko, I. V., Gibney, G. T. & Smalley, K. S. M. NRAS mutant melanoma: biological behavior and future strategies for therapeutic management. Oncogene 32, 3009–3018 (2013).
3. Garnett, M. J. & Marais, R. Guilty as charged: B-RAF is a human oncogene. Cancer Cell 6, 313–319 (2004).
4. Chapman, P. B. et al. Improved survival with vemurafenib in melanoma with BRAF V600E mutation. N. Engl. J. Med. 364, 2507–2516 (2011).
5. Hauschild, A. et al. Dabrafenib in BRAF-mutated metastatic melanoma: a multicentre, open-label, phase 3 randomised controlled trial. Lancet 380, 358–365 (2012).
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