DOT1L safeguards cartilage homeostasis and protects against osteoarthritis-Reference-Cited by-同舟云学术

DOT1L safeguards cartilage homeostasis and protects against osteoarthritis

Published:2017-06-19 Issue:1 Volume:8 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Monteagudo Silvia,Cornelis Frederique M. F.,Aznar-Lopez Carolina,Yibmantasiri Ploi,Guns Laura-An,Carmeliet Peter,Cailotto Frédéric,Lories Rik J.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms15889.pdf

Reference59 articles.

1. Bijlsma, J. W. J., Berenbaum, F. & Lafeber, F. P. J. G. Osteoarthritis: an update with relevance for clinical practice. Lancet 377, 2115–2126 (2011).

2. Conaghan, P. G., Kloppenburg, M., Schett, G. & Bijlsma, J. W. J. EULAR osteoarthritis ad hoc committee. Osteoarthritis research priorities: a report from a EULAR ad hoc expert committee. Ann. Rheum. Dis. 73, 1442–1445 (2014).

3. Steger, D. J. et al. DOT1L/KMT4 recruitment and H3K79 methylation are ubiquitously coupled with gene transcription in mammalian cells. Mol. Cell Biol. 28, 2825–2839 (2008).

4. Nguyen, A. T. & Zhang, Y. The diverse functions of Dot1 and H3K79 methylation. Genes Dev. 25, 1345–1358 (2011).

5. Feng, Q. et al. Methylation of H3-Lysine 79 is mediated by a new family of HMTases without a SET domain. Curr. Biol. 12, 1052–1058 (2002).

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