TP53 mutations and CDKN2A mutations/deletions are highly recurrent molecular alterations in the malignant progression of sinonasal papillomas-Reference-Cited by-同舟云学术

TP53 mutations and CDKN2A mutations/deletions are highly recurrent molecular alterations in the malignant progression of sinonasal papillomas

Published:2020-11-17 Issue:6 Volume:34 Page:1133-1142
ISSN:0893-3952
Container-title:Modern Pathology
language:en
Short-container-title:Mod Pathol

Author:

Brown Noah A.,Plouffe Komal R.,Yilmaz Osman,Weindorf Steven C.,Betz Bryan L.,Carey Thomas E.,Seethala Raja R.,McHugh Jonathan B.,Tomlins Scott A.,Udager Aaron M.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Pathology and Forensic Medicine

Link

http://www.nature.com/articles/s41379-020-00716-3.pdf

Reference22 articles.

1. Barnes L. Schneiderian papillomas and nonsalivary glandular neoplasms of the head and neck. Mod Pathol. 2002;15:279–97.

2. Weindorf SC, Brown NA, McHugh JB, Udager AM. Sinonasal papillomas and carcinomas a contemporary update with review of an emerging molecular classification. Arch Pathol Lab Med. 2019;143:1304–16.

3. Udager AM, Rolland DCM, McHugh JB, Betz BL, Murga-Zamalloa C, Carey TE, et al. High-frequency targetable EGFR mutations in sinonasal squamous cell carcinomas arising from inverted sinonasal papilloma. Cancer Res. 2015;75:2600–6.

4. Udager AM, McHugh JB, Betz BL, Montone KT, Livolsi VA, Seethala RR, et al. Activating KRAS mutations are characteristic of oncocytic sinonasal papilloma and associated sinonasal squamous cell carcinoma. J Pathol. 2016;239:394–8.

5. Udager AM, McHugh JB, Goudsmit CM, Weigelin HC, Lim MS, Elenitoba-Johnson KSJ, et al. Human papillomavirus (HPV) and somatic EGFR mutations are essential, mutually exclusive oncogenic mechanisms for inverted sinonasal papillomas and associated sinonasal squamous cell carcinomas. Ann Oncol. 2018;29:466–71.

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