ASIC1 and ASIC3 contribute to acidity-induced EMT of pancreatic cancer through activating Ca2+/RhoA pathway-Reference-Cited by-同舟云学术

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ASIC1 and ASIC3 contribute to acidity-induced EMT of pancreatic cancer through activating Ca2+/RhoA pathway

Published:2017-05 Issue:5 Volume:8 Page:e2806-e2806
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Zhu Shuai,Zhou Hai-Yun,Deng Shi-Chang,Deng Shi-Jiang,He Chi,Li Xiang,Chen Jing-Yuan,Jin Yan,Hu Zhuang-Li,Wang Fang,Wang Chun-You,Zhao Gang

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2017189.pdf

Reference48 articles.

1. Hidalgo M . Pancreatic cancer. N Engl J Med 2010; 362: 1605–1617.

2. Haeno H, Gonen M, Davis MB, Herman JM, Iacobuzio-Donahue CA, Michor F . Computational modeling of pancreatic cancer reveals kinetics of metastasis suggesting optimum treatment strategies. Cell 2012; 148: 362–375.

3. Tuveson DA, Neoptolemos JP . Understanding metastasis in pancreatic cancer: a call for new clinical approaches. Cell 2012; 148: 21–23.

4. Polyak K, Weinberg RA . Transitions between epithelial and mesenchymal states: acquisition of malignant and stem cell traits. Nat Rev Cancer 2009; 9: 265–273.

5. Javle MM, Gibbs JF, Iwata KK, Pak Y, Rutledge P, Yu J et al. Epithelial-mesenchymal transition (EMT) and activated extracellular signal-regulated kinase (p-Erk) in surgically resected pancreatic cancer. Ann Surg Oncol 2007; 14: 3527–3533.

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