PINK1-induced mitophagy promotes neuroprotection in Huntington’s disease-Reference-Cited by-同舟云学术

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PINK1-induced mitophagy promotes neuroprotection in Huntington’s disease

Published:2015-01 Issue:1 Volume:6 Page:e1617-e1617
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Khalil B,El Fissi N,Aouane A,Cabirol-Pol M-J,Rival T,Liévens J-C

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2014581.pdf

Reference37 articles.

1. Browne SE, Bowling AC, MacGarvey U, Baik MJ, Berger SC, Muqit MM et al. Oxidative damage and metabolic dysfunction in Huntington’s disease: selective vulnerability of the basal ganglia. Ann Neurol 1997; 41: 646–653.

2. Gu M, Gash MT, Mann VM, Javoy-Agid F, Cooper JM, Schapira AH . Mitochondrial defect in Huntington’s disease caudate nucleus. Ann Neurol 1996; 39: 385–389.

3. Cui L, Jeong H, Borovecki F, Parkhurst CN, Tanese N, Krainc D . Transcriptional repression of PGC-1alpha by mutant huntingtin leads to mitochondrial dysfunction and neurodegeneration. Cell 2006; 127: 59–69.

4. Kim J, Moody JP, Edgerly CK, Bordiuk OL, Cormier K, Smith K et al. Mitochondrial loss, dysfunction and altered dynamics in Huntington’s disease. Hum Mol Genet 2010; 19: 3919–3935.

5. Shirendeb UP, Calkins MJ, Manczak M, Anekonda V, Dufour B, McBride JL et al. Mutant huntingtin’s interaction with mitochondrial protein Drp1 impairs mitochondrial biogenesis and causes defective axonal transport and synaptic degeneration in Huntington’s disease. Hum Mol Genet 2012; 21: 406–420.

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