The bromodomain protein BRD4 regulates the KEAP1/NRF2-dependent oxidative stress response-Reference-Cited by-同舟云学术

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The bromodomain protein BRD4 regulates the KEAP1/NRF2-dependent oxidative stress response

Published:2014-04 Issue:4 Volume:5 Page:e1195-e1195
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Hussong M,Börno S T,Kerick M,Wunderlich A,Franz A,Sültmann H,Timmermann B,Lehrach H,Hirsch-Kauffmann M,Schweiger M R

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2014157.pdf

Reference59 articles.

1. Schreck R, Rieber P, Baeuerle PA . Reactive oxygen intermediates as apparently widely used messengers in the activation of the NF-kappa B transcription factor and HIV-1. EMBO J 1991; 10: 2247–2258.

2. McMahon M, Itoh K, Yamamoto M, Hayes JD . Keap1-dependent proteasomal degradation of transcription factor Nrf2 contributes to the negative regulation of antioxidant response element-driven gene expression. J Biol Chem 2003; 278: 21592–21600.

3. Alam J, Cook JL . Transcriptional regulation of the heme oxygenase-1 gene via the stress response element pathway. Curr Pharm Des 2003; 9: 2499–2511.

4. Kobayashi A, Ohta T, Yamamoto M . Unique function of the Nrf2-Keap1 pathway in the inducible expression of antioxidant and detoxifying enzymes. Methods Enzymol 2004; 378: 273–286.

5. Itoh K, Wakabayashi N, Katoh Y, Ishii T, O'Connor T, Yamamoto M . Keap1 regulates both cytoplasmic-nuclear shuttling and degradation of Nrf2 in response to electrophiles. Genes Cells 2003; 8: 379–391.

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