Impairment of enzymatic antioxidant defenses is associated with bilirubin-induced neuronal cell death in the cerebellum of Ugt1 KO mice-Reference-Cited by-同舟云学术

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Impairment of enzymatic antioxidant defenses is associated with bilirubin-induced neuronal cell death in the cerebellum of Ugt1 KO mice

Published:2015-05 Issue:5 Volume:6 Page:e1739-e1739
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Bortolussi G,Codarin E,Antoniali G,Vascotto C,Vodret S,Arena S,Cesaratto L,Scaloni A,Tell G,Muro A F

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2015113.pdf

Reference69 articles.

1. Stocker R, Yamamoto Y, McDonagh AF, Glazer AN, Ames BN . Bilirubin is an antioxidant of possible physiological importance. Science 1987; 235: 1043–1046.

2. Shapiro SM . Bilirubin toxicity in the developing nervous system. Pediatr Neurol 2003; 29: 410–421.

3. Seppen J, Bosma PJ, Goldhoorn BG, Bakker CT, Chowdhury JR, Chowdhury NR et al. Discrimination between Crigler-Najjar type I and II by expression of mutant bilirubin uridine diphosphate-glucuronosyltransferase. J Clin Invest 1994; 94: 2385–2391.

4. Strauss KA, Robinson DL, Vreman HJ, Puffenberger EG, Hart G, Morton DH . Management of hyperbilirubinemia and prevention of kernicterus in 20 patients with Crigler-Najjar disease. Eur J Pediatr 2006; 165: 306–319.

5. Pett S, Mowat AP . Crigler-Najjar syndrome types I and II. Clinical experience—King's College Hospital 1972-1978. Phenobarbitone, phototherapy and liver transplantation. Mol Aspects Med 1987; 9: 473–482.

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