Decreased vitamin B12 availability induces ER stress through impaired SIRT1-deacetylation of HSF1-Reference-Cited by-同舟云学术

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Decreased vitamin B12 availability induces ER stress through impaired SIRT1-deacetylation of HSF1

Published:2013-03 Issue:3 Volume:4 Page:e553-e553
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Ghemrawi R,Pooya S,Lorentz S,Gauchotte G,Arnold C,Gueant J-L,Battaglia-Hsu S-F

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis201369.pdf

Reference47 articles.

1. Spriggs KA, Bushell M, Willis AE . Translational regulation of gene expression during conditions of cell stress. Mol Cell 2010; 40: 228–237.

2. Bhate V, Deshpande S, Bhat D, Joshi N, Ladkat R, Watve S et al. Vitamin B12 status of pregnant Indian women and cognitive function in their 9-year-old children. Food Nutr Bull 2008; 29: 249–254.

3. Garrod MG, Green R, Allen LH, Mungas DM, Jagust WJ, Haan MN et al. Fraction of total plasma vitamin B12 bound to transcobalamin correlates with cognitive function in elderly Latinos with depressive symptoms. Clin Chem 2008; 54: 1210–1217.

4. Hooshmand B, Solomon A, Kåreholt I, Leiviskä J, Rusanen M, Ahtiluoto S et al. Homocysteine and holotranscobalamin and the risk of Alzheimer disease: a longitudinal study. Neurology 2010; 75: 1408–1414.

5. McCaddon A . Homocysteine and cognition--a historical perspective. J Alzheimers Dis 2006; 9: 361–380.

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