Antitumor activity of IL-32β through the activation of lymphocytes, and the inactivation of NF-κB and STAT3 signals-Reference-Cited by-同舟云学术

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Antitumor activity of IL-32β through the activation of lymphocytes, and the inactivation of NF-κB and STAT3 signals

Published:2013-05 Issue:5 Volume:4 Page:e640-e640
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Yun H-M,Oh J H,Shim J-H,Ban J O,Park K-R,Kim J-H,Lee D H,Kang J-W,Park Y H,Yu D,Kim Y,Han S B,Yoon D-Y,Hong J T

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2013166.pdf

Reference56 articles.

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2. Lewis CE, Pollard JW . Distinct role of macrophages in different tumor microenvironments. Cancer Res 2006; 66: 605–612.

3. Lin WW, Karin M . A cytokine-mediated link between innate immunity, inflammation, and cancer. J Clin Invest 2007; 117: 1175–1183.

4. Jee SH, Shen SC, Chiu HC, Tsai WL, Kuo ML . Overexpression of interleukin-6 in human basal cell carcinoma cell lines increases anti-apoptotic activity and tumorigenic potency. Oncogene 2001; 20: 198–208.

5. Ishiguro H, Akimoto K, Nagashima Y, Kojima Y, Sasaki T, Ishiguro-Imagawa Y et al. aPKClambda/iota promotes growth of prostate cancer cells in an autocrine manner through transcriptional activation of interleukin-6. Proc Natl Acad Sci USA 2009; 106: 16369–16374.

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