DNA damage induces reactive oxygen species generation through the H2AX-Nox1/Rac1 pathway-Reference-Cited by-同舟云学术

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DNA damage induces reactive oxygen species generation through the H2AX-Nox1/Rac1 pathway

Published:2012-01 Issue:1 Volume:3 Page:e249-e249
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Kang M A,So E-Y,Simons A L,Spitz D R,Ouchi T

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2011134.pdf

Reference39 articles.

1. Bassing CH, Alt FW . The cellular response to general and programmed DNA double strand breaks. DNA Repair 2004; 3: 781–796.

2. Zhou BB, Elledge SJ . The DNA damage response: putting checkpoints in perspective. Nature 2000; 408: 433–439.

3. Ciccia A, Elledge SJ . The DNA damage response: making it safe to play with knives. Mol Cell 2010; 40: 179–204.

4. Fernandez-Capetillo O, Lee A, Nussenzweig M, Nussenzweig A . H2AX: the histone guardian of the genome. DNA Repair 2004; 3: 959–967.

5. Stucki M, Clapperton JA, Mohammad D, Yaffe MB, Smerdon SJ, Jackson SP . MDC1 directly binds phosphorylated histone H2AX to regulate cellular responses to DNA double-strand breaks. Cell 2005; 123: 1213–1226.

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