Interaction between autophagy and senescence is required for dihydroartemisinin to alleviate liver fibrosis-Reference-Cited by-同舟云学术

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Interaction between autophagy and senescence is required for dihydroartemisinin to alleviate liver fibrosis

Published:2017-06 Issue:6 Volume:8 Page:e2886-e2886
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Zhang Zili,Yao Zhen,Zhao Shifeng,Shao Jiangjuan,Chen Anping,Zhang Feng,Zheng Shizhong

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2017255.pdf

Reference46 articles.

1. Lee YA, Wallace MC, Friedman SL . Pathobiology of liver fibrosis: a translational success story. Gut 2015; 64: 830–841.

2. Kang N, Gores GJ, Shah VH . Hepatic stellate cells: partners in crime for liver metastases? Hepatology 2011; 54: 707–713.

3. Kitano M, Bloomston PM . Hepatic stellate cells and microRNAs in pathogenesis of liver fibrosis. J Clin Med 2016; 5: 10–13.

4. Lambrecht J, Mannaerts I, van Grunsven LA . The role of miRNAs in stress-responsive hepatic stellate cells during liver fibrosis. Front Physiol 2015; 6: 209.

5. Thompson AI, Conroy KP, Henderson NC . Hepatic stellate cells: central modulators of hepatic carcinogenesis. BMC Gastroenterol 2015; 15: 63.

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4. The transcription factor GATA6 accelerates vascular smooth muscle cell senescence-related arterial calcification by counteracting the role of anti-aging factor SIRT6 and impeding DNA damage repair;Kidney International;2024-01

5. Dihydroartemisinin attenuates ischemia/reperfusion-induced renal tubular senescence by activating autophagy;Chinese Journal of Natural Medicines;2023-09

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