p57KIP2 control of actin cytoskeleton dynamics is responsible for its mitochondrial pro-apoptotic effect

Author:

Kavanagh E,Vlachos P,Emourgeon V,Rodhe J,Joseph B

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Reference36 articles.

1. Kavanagh E, Joseph B . The hallmarks of CDKN1C (p57, KIP2) in cancer. Biochim Biophys Acta 2011; 1816: 50–56.

2. Lam WW, Hatada I, Ohishi S, Mukai T, Joyce JA, Cole TR et al. Analysis of germline CDKN1C (p57KIP2) mutations in familial and sporadic Beckwith-Wiedemann syndrome (BWS) provides a novel genotype-phenotype correlation. J Med Genet 1999; 36: 518–523.

3. Oya M, Schulz WA . Decreased expression of p57(KIP2)mRNA in human bladder cancer. Br J Cancer 2000; 83: 626–631.

4. Li JQ, Wu F, Usuki H, Kubo A, Masaki T, Fujita J et al. Loss of p57KIP2 is associated with colorectal carcinogenesis. Int J Oncol 2003; 23: 1537–1543.

5. Larson PS, Schlechter BL, King CL, Yang Q, Glass CN, Mack C et al. CDKN1C/p57kip2 is a candidate tumor suppressor gene in human breast cancer. BMC Cancer 2008; 8: 68.

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