Trehalose ameliorates oxidative stress-mediated mitochondrial dysfunction and ER stress via selective autophagy stimulation and autophagic flux restoration in osteoarthritis development-Reference-Cited by-同舟云学术

Trehalose ameliorates oxidative stress-mediated mitochondrial dysfunction and ER stress via selective autophagy stimulation and autophagic flux restoration in osteoarthritis development

Published:2017-10 Issue:10 Volume:8 Page:e3081-e3081
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Tang Qian,Zheng Gang,Feng Zhenhua,Chen Yu,Lou Yiting,Wang Chenggui,Zhang Xiaolei,Zhang Yu,Xu Huazi,Shang Ping,Liu Haixiao^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2017453.pdf

Reference54 articles.

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2. Karsdal MA, Michaelis M, Ladel C, Siebuhr AS, Bihlet AR, Andersen JR et al. Disease-modifying treatments for osteoarthritis (DMOADs) of the knee and hip: lessons learned from failures and opportunities for the future. Osteoarthritis Cartilage 2016; 24: 2013–2021.

3. Charlier E, Relic B, Deroyer C, Malaise O, Neuville S, Collee J et al. Insights on molecular mechanisms of chondrocytes death in osteoarthritis. Int J Mol Sci 2016; 17: E2146.

4. Lepetsos P, Papavassiliou AG . ROS/oxidative stress signaling in osteoarthritis. Biochim Biophys Acta 2016; 1862: 576–591.

5. Li D, Xie G, Wang W . Reactive oxygen species: the 2-edged sword of osteoarthritis. Am J Med Sci 2012; 344: 486–490.

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