HDAC8 and STAT3 repress BMF gene activity in colon cancer cells-Reference-Cited by-同舟云学术

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HDAC8 and STAT3 repress BMF gene activity in colon cancer cells

Published:2014-10 Issue:10 Volume:5 Page:e1476-e1476
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Kang Y,Nian H,Rajendran P,Kim E,Dashwood W M,Pinto J T,Boardman L A,Thibodeau S N,Limburg P J,Löhr C V,Bisson W H,Williams D E,Ho E,Dashwood R H

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2014422.pdf

Reference55 articles.

1. Campbell RM, Tummino PJ . Cancer epigenetics drug discovery and development: the challenge of hitting the mark. J Clin Invest 2014; 124: 64–69.

2. Marks PA, Wu WS . Histone deacetylase inhibitors: potential in cancer therapy. J Cell Biochem 2009; 107: 600–608.

3. Lee JH, Choy ML, Marks PA . Mechanisms of resistance to histone deacetylase inhibitors. Adv Cancer Res 2012; 116: 39–86.

4. Rajendran P, Williams DE, Ho E, Dashwood RH . Metabolism as a key to histone deacetylase inhibition. Crit Rev Biochem Mol Biol 2011; 46: 181–199.

5. Bishton MJ, Harrison SJ, Martin BP, McLaughlin N, James C, Josefsson EC et al. Deciphering the molecular and biological processes that mediate histone deacetylase inhibitor-induced thrombocytopenia. Blood 2011; 117: 3658–3668.

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