Endoplasmic reticulum stress inhibition protects steatotic and non-steatotic livers in partial hepatectomy under ischemia–reperfusion-Reference-Cited by-同舟云学术

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Endoplasmic reticulum stress inhibition protects steatotic and non-steatotic livers in partial hepatectomy under ischemia–reperfusion

Published:2010-07 Issue:7 Volume:1 Page:e52-e52
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Mosbah I Ben,Alfany-Fernández I,Martel C,Zaouali M A,Bintanel-Morcillo M,Rimola A,Rodés J,Brenner C,Roselló-Catafau J,Peralta C

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis201029.pdf

Reference36 articles.

1. Ramalho FS, Alfany-Fernandez I, Casillas-Ramirez A, Massip-Salcedo M, Serafin A, Rimola A et al. Are angiotensin II receptor antagonists useful strategies in steatotic and nonsteatotic livers in conditions of partial hepatectomy under ischemia-reperfusion? J Pharmacol Exp Ther 2009; 329: 130–140.

2. Kammoun HL, Chabanon H, Hainault I, Luquet S, Magnan C, Koike T et al. GRP78 expression inhibits insulin and ER stress-induced SREBP-1c activation and reduces hepatic steatosis in mice. J Clin Invest 2009; 119: 1201–1215.

3. Xu C, Bailly-Maitre B, Reed JC . Endoplasmic reticulum stress: cell life and death decisions. J Clin Invest 2005; 115: 2656–2664.

4. Ozcan U, Yilmaz E, Ozcan L, Furuhashi M, Vaillancourt E, Smith RO et al. Chemical chaperones reduce ER stress and restore glucose homeostasis in a mouse model of type 2 diabetes. Science 2006; 313: 1137–1140.

5. Yoneda T, Imaizumi K, Oono K, Yui D, Gomi F, Katayama T et al. Activation of caspase-12, an endoplastic reticulum (ER) resident caspase, through tumor necrosis factor receptor-associated factor 2-dependent mechanism in response to the ER stress. J Biol Chem 2001; 276: 13935–13940.

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