lncRNA MIAT functions as a competing endogenous RNA to upregulate DAPK2 by sponging miR-22-3p in diabetic cardiomyopathy-Reference-Cited by-同舟云学术

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lncRNA MIAT functions as a competing endogenous RNA to upregulate DAPK2 by sponging miR-22-3p in diabetic cardiomyopathy

Published:2017-07 Issue:7 Volume:8 Page:e2929-e2929
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Zhou Xiang,Zhang Wei,Jin Mengchao,Chen Jianchang,Xu Weiting,Kong Xiangqing

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis2017321.pdf

Reference18 articles.

1. Aneja A, Tang WH, Bansilal S, Garcia MJ, Farkouh ME . Diabetic cardiomyopathy: insights into pathogenesis, diagnostic challenges, and therapeutic options. Am J Med 2008; 121: 748–757.

2. Bugger H, Abel ED . Molecular mechanisms of diabetic cardiomyopathy. Diabetologia 2014; 57: 660–671.

3. Moran VA, Perera RJ, Khalil AM . Emerging functional and mechanistic paradigms of mammalian long non-coding RNAs. Nucleic Acids Res 2012; 40: 6391–6400.

4. I Ishii N, Ozaki K, Sato H, Mizuno H, Saito S, Takahashi A et al. Identification of a novel non-coding RNA, MIAT, that confers risk of myocardial infarction. J Hum Genet 2006; 51: 1087–1099.

5. Yan B, Yao J, Liu JY, Li XM, Wang XQ, Li YJ et al. lncRNA-MIAT regulates microvascular dysfunction by functioning as a competing endogenous RNA. Circ Res 2015; 116: 1143–1156.

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