Dietary zinc supplementation of 3xTg-AD mice increases BDNF levels and prevents cognitive deficits as well as mitochondrial dysfunction-Reference-Cited by-同舟云学术

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Dietary zinc supplementation of 3xTg-AD mice increases BDNF levels and prevents cognitive deficits as well as mitochondrial dysfunction

Published:2010-10 Issue:10 Volume:1 Page:e91-e91
ISSN:2041-4889
Container-title:Cell Death & Disease
language:en
Short-container-title:Cell Death Dis

Author:

Corona C,Masciopinto F,Silvestri E,Viscovo A Del,Lattanzio R,Sorda R La,Ciavardelli D,Goglia F,Piantelli M,Canzoniero L M T,Sensi S L

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Cell Biology,Cellular and Molecular Neuroscience,Immunology

Link

http://www.nature.com/articles/cddis201073.pdf

Reference40 articles.

1. Zatta P, Drago D, Bolognin S, Sensi SL . Alzheimer's disease, metal ions and metal homeostatic therapy. Trends Pharmacol Sci 2009; 30: 346–355.

2. Sensi SL, Paoletti P, Bush AI, Sekler I . Zinc in the physiology and pathology of the CNS. Nat Rev Neurosci 2009; 10: 780–791.

3. Boom A, Authelet M, Dedecker R, Frederick C, Van Heurck R, Daubie V et al. Bimodal modulation of tau protein phosphorylation and conformation by extracellular Zn2+ in human-tau transfected cells. Biochim Biophys Acta 2009; 1793: 1058–1067.

4. Mo ZY, Zhu YZ, Zhu HL, Fan JB, Chen J, Liang Y . Low micromolar zinc accelerates the fibrillization of human tau via bridging of Cys-291 and Cys-322. J Biol Chem 2009; 284: 34648–34657.

5. Arispe N, Pollard HB, Rojas E . Zn2+ interaction with Alzheimer amyloid beta protein calcium channels. Proc Natl Acad Sci USA 1996; 93: 1710–1715.

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