Orphan receptor-GPR52 inverse agonist efficacy in ameliorating chronic stress-related deficits in reward motivation and phasic accumbal dopamine activity in mice-Reference-Cited by-同舟云学术

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Orphan receptor-GPR52 inverse agonist efficacy in ameliorating chronic stress-related deficits in reward motivation and phasic accumbal dopamine activity in mice

Published:2024-09-07 Issue:1 Volume:14 Page:
ISSN:2158-3188
Container-title:Translational Psychiatry
language:en
Short-container-title:Transl Psychiatry

Author:

Zhang Chenfeng,Kúkeľová Diana,Sigrist Hannes,Hengerer Bastian^ORCID,Kratzer Ramona F.,Mracek Philipp,Omrani Azar,von Heimendahl Moritz^ORCID,Pryce Christopher R.^ORCID

Funder

Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung

Boehringer Ingelheim

China Scholarship Council

Publisher

Springer Science and Business Media LLC

Link

https://www.nature.com/articles/s41398-024-03081-w.pdf

Reference53 articles.

1. Admon R, Pizzagalli DA. Dysfunctional reward processing in depression. Curr Opin Psychol. 2015;4:114–8.

2. Husain M, Roiser JP. Neuroscience of apathy and anhedonia: a transdiagnostic approach. Nat Rev Neurosci. 2018;19:470–84.

3. Pizzagalli DA. Depression, stress, and anhedonia: toward a synthesis and integrated model. Annu Rev Clin Psychol. 2014;10:393–423.

4. Kaiser S, Lyne J, Agartz I, Clarke M, Mørch-Johnsen L, Faerden A. Individual negative symptoms and domains—relevance for assessment, pathomechanisms and treatment. Schizophr Res. 2017;186:39–45.

5. Pelizza L, Ferrari A. Anhedonia in schizophrenia and major depression: state or trait? Ann Gen Psychiatry. 2009;8:22.

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