BARMR1-mediated sorafenib resistance is derived through stem-like property acquisition by activating integrin-FAK signaling pathways-Reference-Cited by-同舟云学术

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BARMR1-mediated sorafenib resistance is derived through stem-like property acquisition by activating integrin-FAK signaling pathways

Published:2020-06-12 Issue:1 Volume:5 Page:
ISSN:2059-3635
Container-title:Signal Transduction and Targeted Therapy
language:en
Short-container-title:Sig Transduct Target Ther

Author:

Guo Xing Rong,Shan Meng Ye,Huang Yu,Zhang Zong Li,Yuan Yue,Dai Long Jun,Wang Jue,Zhou Xue Peng,Ji Fu Yun,Tang Jun Ming,Meng Zhong Ji,Ruan Xu Zhi

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics

Link

http://www.nature.com/articles/s41392-020-0189-8.pdf

Reference5 articles.

1. Strausberg, R. L. et al. Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. Proc. Natl Acad. Sci. USA99, 16899–16903 (2002).

2. Ruan, X. Z. et al. Identification and characterization of two novel variants of the DUF1208 protein FAM92A1. Mol. Cells23, 391–397 (2007).

3. Wang, L. et al. FAM92A1 is a BAR domain protein required for mitochondrial ultrastructure and function. J. Cell Biol.218, 97–111 (2019).

4. Liang, S. et al. Prokaryotic expression, purification of a new tumor-relative protein FAM92A1-289 and its characterization in renal cell carcinoma. Cancer Lett.276, 81–87 (2009).

5. Guo, X. R. et al. Nuclear FAM289-Galectin-1 interaction controls FAM289-mediated tumor promotion in malignant glioma. J. Exp. Clin. Cancer Res.38, 394–433 (2019).

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