Calreticulin surface presentation: a signal for natural killer cells to attack
Author:
Funder
U.S. Department of Health & Human Services | NIH | National Institute of General Medical Sciences
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics
Link
https://www.nature.com/articles/s41392-023-01551-z.pdf
Reference5 articles.
1. Sen Santara, S. et al. The NK cell receptor NKp46 recognizes ecto-calreticulin on ER-stressed cells. Nature 616, 348–356 (2023).
2. Chao, M. P. et al. Calreticulin is the dominant pro-phagocytic signal on multiple human cancers and is counterbalanced by CD47. Sci. Transl. Med. 2, 63ra94 (2010).
3. Shoulders, M. D. et al. Stress-independent activation of XBP1s and/or ATF6 reveals three functionally diverse ER proteostasis environments. Cell Rep. 3, 1279–1292 (2013).
4. Van Krieken, R., Tsai, Y.-L., Carlos, A. J., Ha, D. P. & Lee, A. S. ER residential chaperone GRP78 unconventionally relocalizes to the cell surface via endosomal transport. Cell. Mol. Life Sci. 78, 5179–5195 (2021).
5. Zhang, M. et al. Calreticulin as a marker and therapeutic target for cancer. Clin. Exp. Med. https://doi.org/10.1007/s10238-022-00937-7 (2022).
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