Berbamine inhibits SARS-CoV-2 infection by compromising TRPMLs-mediated endolysosomal trafficking of ACE2-Reference-Cited by-同舟云学术

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Berbamine inhibits SARS-CoV-2 infection by compromising TRPMLs-mediated endolysosomal trafficking of ACE2

Published:2021-04-24 Issue:1 Volume:6 Page:
ISSN:2059-3635
Container-title:Signal Transduction and Targeted Therapy
language:en
Short-container-title:Sig Transduct Target Ther

Author:

Huang Lihong^ORCID,Yuen Terrence Tsz-Tai,Ye Zuodong^ORCID,Liu Shuyan,Zhang Guoliang^ORCID,Chu Hin,Yue Jianbo^ORCID

Funder

National Science Foundation of China | National Natural Science Foundation of China-Yunnan Joint Fund

Research Grants Council, University Grants Committee

Shenzhen Science and Technology Innovation Commission

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics

Link

http://www.nature.com/articles/s41392-021-00584-6.pdf

Reference5 articles.

1. Letko, M., Marzi, A. & Munster, V. Functional assessment of cell entry and receptor usage for SARS-CoV-2 and other lineage B betacoronaviruses. Nat. Microbiol. 5, 562–569 (2020).

2. Zhou, Y., Frey, T. K. & Yang, J. J. Viral calciomics: interplays between Ca2+ and virus. Cell Calcium 46, 1–17 (2009).

3. Zhang, C. M., Gao, L., Zheng, Y. J. & Yang, H. T. Berbamine protects the heart from ischemia/reperfusion injury by maintaining cytosolic Ca(2+) homeostasis and preventing calpain activation. Circulation J. 76, 1993–2002 (2012).

4. Venkatachalam, K., Wong, C.-O. & Zhu, M. X. The role of TRPMLs in endolysosomal trafficking and function. Cell Calcium 58, 48–56 (2015).

5. Ortega, F. G. et al. Interfering with endolysosomal trafficking enhances release of bioactive exosomes. Nanomedicine 20, 102014 (2019).

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