Mechanisms of the thapsigargin-induced Ca2+entry inin situendothelial cells of the porcine aortic valve and the endothelium-dependent relaxation in the porcine coronary artery
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1038/sj.bjp.0703548/fullpdf
Reference50 articles.
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3. Sensitivity of G-protein involved in endothelin-1-induced Ca2+ influx to pertussis toxin in porcine endothelial cells in situ;AOKI;Br. J. Pharmacol.,1994
4. Endothelin induces the Ca2+-transient in endothelial cells in situ;AOKI;Biochem. Biophys. Res. Commun.,1991
5. Inhibition of thapsigargin-induced calcium entry by microinjected guanine nucleotide analogues. Evidence for the involvement of a small G-protein in capacitative calcium entry;BIRD;J. Biol. Chem.,1993
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