Left regional cardiac perfusion in vitro with platelet-activating factor, norepinephrine and K+ reveals that ischaemic arrhythmias are caused by independent effects of endogenous ‘mediators’ facilitated by interactions, and moderated by paradoxical antago

Author:

Baker Kathryn E,Curtis Michael J

Publisher

Wiley

Subject

Pharmacology

Reference65 articles.

1. Human and rabbit platelets form platelet activating factor in response to calcium ionophore;ALAM;Thrombosis Res.,1983

2. Thromboxane and prostacyclin release during cardiac immediate hypersensitivity reactions in vitro;ALLAN;J. Pharmacol. Exp. Therap.,1981

3. Norepinephrine-induced relaxations in rat aorta mediated by endothelial beta-adrenoceptors - impairment by aging and hypertension;ARRIBAS;J. Pharmacol. Exp. Therap.,1994

4. Independent dual perfusion of left and right coronary arteries in isolated rat hearts;AVKIRAN;Am. J. Physiol.,1991

5. Protection against ventricular fibrillation by the PAF antagonist, BN-50739, involves an ischaemia-selective mechanism;BAKER;J. Cardiovasc. Pharmacol.,1999

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