Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action-Reference-Cited by-同舟云学术

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Blockade of HERG channels by the class III antiarrhythmic azimilide: mode of action

Published:1998-01 Issue:1 Volume:123 Page:23-30
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:British Journal of Pharmacology

Author:

Busch A E¹²,Eigenberger B¹,Jurkiewicz N K³,Salata J J³,Pica A⁴,Suessbrich H¹,Lang F¹

Affiliation:

1. Institute of Physiology I, Eberhard‐Karls University of Tübingen, Gmelinstr. 5, 72076 Tübingen

2. Max‐Planck‐Institute for Experimental Medicine, Hermann‐Rein‐Str. 3, 37075 Göttingen, Germany

3. Department of Pharmacology, Merck Research Laboratories, West Point, PA 19486‐0004, U.S.A.

4. Seconda Universita Degli Studi di Napoli, Cattedra di Nefrologia, Via S. Pansini 5, Padiglione 17, 80131 Napoli, Italy

Publisher

Wiley

Subject

Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1038/sj.bjp.0701575

Reference37 articles.

1. KvLQT1 and IsK (minK) proteins associate to form the IKS cardiac potassium current

2. Protection Against Programmed Electrical Stimulation-Induced Ventricular Tachycardia and Sudden Cardiac Death by NE-10064, a Class III Antiarrhythmic Drug

3. Efficacy of the Class III Antiarrhythmic Agent Azimilide in Rodent Models of Ventricular Arrhythmia

4. Blockade of human IsK channels expressed in Xenopus oocytes by the novel class III antiarrhythmic NE-10064

5. Effect of isosorbiddinitrate on exogenously expressed slowly activating K+ channels and endogenous K+ channels in Xenopus oocytes.

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