A Functional Polymorphism (rs2494752) in the AKT1 Promoter Region and Gastric Adenocarcinoma Risk in an Eastern Chinese Population-Reference-Cited by-同舟云学术

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A Functional Polymorphism (rs2494752) in the AKT1 Promoter Region and Gastric Adenocarcinoma Risk in an Eastern Chinese Population

Published:2016-01-28 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Wang Meng-Yun,He Jing,Zhu Mei-Ling,Teng Xiao-Yan,Li Qiao-Xin,Sun Meng-Hong,Wang Xiao-Feng,Yang Ya-Jun,Wang Jiu-Cun,Jin Li,Wang Ya-Nong,Wei Qing-Yi

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep20008.pdf

Reference35 articles.

1. Torre, L. A. et al. Global cancer statistics, 2012. CA Cancer J Clin 65, 87–108 (2015).

2. Downward, J. PI 3-kinase, Akt and cell survival. Semin Cell Dev Biol 15, 177–82 (2004).

3. Testa, J. R. & Bellacosa, A. AKT plays a central role in tumorigenesis. Proc Natl Acad Sci USA 98, 10983–5 (2001).

4. Staal, S. P. Molecular cloning of the akt oncogene and its human homologues AKT1 and AKT2: amplification of AKT1 in a primary human gastric adenocarcinoma. Proc Natl Acad Sci USA 84, 5034–7 (1987).

5. Cheng, J. Q. et al. AKT2, a putative oncogene encoding a member of a subfamily of protein-serine/threonine kinases, is amplified in human ovarian carcinomas. Proc Natl Acad Sci USA 89, 9267–71 (1992).

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