Alcohol reduces muscle fatigue through atomistic interactions with nicotinic receptors-Reference-Cited by-同舟云学术

Alcohol reduces muscle fatigue through atomistic interactions with nicotinic receptors

Published:2018-10-03 Issue:1 Volume:1 Page:
ISSN:2399-3642
Container-title:Communications Biology
language:en
Short-container-title:Commun Biol

Author:

Noori Hamid R.,Mücksch Christian,Vengeliene Valentina,Schönig Kai,Takahashi Tatiane T.,Mukhtasimova Nuriya,Bagher Oskouei Maryam,Mosqueira Matias,Bartsch Dusan,Fink Rainer,Urbassek Herbert M.,Spanagel Rainer,Sine Steven M.

Funder

Bundesministerium für Bildung und Forschung

EC | Horizon 2020

Publisher

Springer Science and Business Media LLC

Subject

General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology,Medicine (miscellaneous)

Link

http://www.nature.com/articles/s42003-018-0157-9.pdf

Reference70 articles.

1. Spanagel, R. Alcoholism: a systems approach from molecular physiology to addictive behavior. Physiol. Rev. 89, 649–705 (2009).

2. Lovinger, D. M., White, G. & Weight, F. F. Ethanol inhibits NMDA-activated ion current in hippocampal neurons. Science 243, 1721–1724 (1989).

3. Mihic, S. J. et al. Sites of alcohol and volatile anaesthetic action on GABA(A) and glycine receptors. Nature 389, 385–389 (1997).

4. Narahashi, T., Aistrup, G. L., Marszalec, W. & Nagata, K. Neuronal nicotinic acetylcholine receptors: a new target site of ethanol. Neurochem. Int. 35, 131–141 (1999).

5. Sundstrom-Poromaa, I. et al. Hormonally regulated alpha(4)beta(2)delta GABA(A) receptors are a target for alcohol. Nat. Neurosci. 5, 721–722 (2002).

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