Effect of postinjury intravenous or intrathecal methylprednisolone on spinal cord excitatory amino-acid release, nitric oxide generation, PGE2 synthesis, and myeloperoxidase content in a pig model of acute spinal cord injury
Author:
Publisher
Springer Science and Business Media LLC
Subject
Clinical Neurology,Neurology,General Medicine
Link
http://www.nature.com/articles/3101891.pdf
Reference50 articles.
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2. Anderson DK et al. Lipid hydrolysis and peroxidation in injured spinal cord: partial protection with methylprednisolone or vitamin E and selenium. Central Nerv Syst Trauma 1985; 2: 257–267.
3. Saunders RD, Dugan LL, Demediuk P, Means ED, Horrocks LA, Anderson DK . Effects of methylprednisolone and the combination of alpha-tocopherol and selenium on arachidonic acid metabolism and lipid peroxidation in traumatized spinal cord tissue. J Neurochem 1987; 49: 24–31.
4. Liu D, McAdoo DJ . Methylprednisolone reduces excitatory amino acid release following experimental spinal cord injury. Brain Res 1993; 609: 293–297.
5. Farooque M, Hillered L, Holtz A, Olsson Y . Effects of methylprednisolone on extracellular lactic acidosis and amino acids after severe compression injury of rat spinal cord. J Neurochem 1996; 66: 1125–1130.
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