Effect of postinjury intravenous or intrathecal methylprednisolone on spinal cord excitatory amino-acid release, nitric oxide generation, PGE2 synthesis, and myeloperoxidase content in a pig model of acute spinal cord injury-Reference-Cited by-同舟云学术

Effect of postinjury intravenous or intrathecal methylprednisolone on spinal cord excitatory amino-acid release, nitric oxide generation, PGE2 synthesis, and myeloperoxidase content in a pig model of acute spinal cord injury

Published:2006-01-24 Issue:10 Volume:44 Page:594-604
ISSN:1362-4393
Container-title:Spinal Cord
language:en
Short-container-title:Spinal Cord

Author:

Bernards C M,Akers T

Publisher

Springer Science and Business Media LLC

Subject

Clinical Neurology,Neurology,General Medicine

Link

http://www.nature.com/articles/3101891.pdf

Reference50 articles.

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3. Saunders RD, Dugan LL, Demediuk P, Means ED, Horrocks LA, Anderson DK . Effects of methylprednisolone and the combination of alpha-tocopherol and selenium on arachidonic acid metabolism and lipid peroxidation in traumatized spinal cord tissue. J Neurochem 1987; 49: 24–31.

4. Liu D, McAdoo DJ . Methylprednisolone reduces excitatory amino acid release following experimental spinal cord injury. Brain Res 1993; 609: 293–297.

5. Farooque M, Hillered L, Holtz A, Olsson Y . Effects of methylprednisolone on extracellular lactic acidosis and amino acids after severe compression injury of rat spinal cord. J Neurochem 1996; 66: 1125–1130.

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