Paraplegic mice are leading to new advances in spinal cord injury research
Author:
Publisher
Springer Science and Business Media LLC
Subject
Neurology (clinical),Neurology,General Medicine
Link
http://www.nature.com/articles/3101754.pdf
Reference18 articles.
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2. Kwon BK, Tetzlaff W, Grauer JN, Beiner J, Vaccaro AR . Pathophysiology and pharmacologic treatment of acute spinal cord injury. Spine J 2004; 4: 451–464.
3. Park E . The role of excitatoxicity in secondary mechanisms of spinal cord injury: a review with an emphasis on the implications for white matter degeneration. J Neurotrauma 2004; 21: 754–774.
4. Simonen M et al. Systemic deletion of the myelin-associated outgrowth inhibitor Nogo-A improves regenerative and plastic responses after spinal cord injury. Neuron 2003; 38: 201–211.
5. Kim JE, Liu BP, Park JH, Strittmatter SM . Nogo-66 receptor prevents raphespinal and rubrospinal axon regeneration and limits functional recovery from spinal cord injury. Neuron 2004; 44: 439–451.
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