Recurrent vesical calculi, hypercalciuria and biochemical evidence of increased bone resorption in an adult male with paraplegia due to spinal cord injury: is there a role for intermittent oral disodium etidronate therapy for prevention of calcium phosphate bladder stones?

Author:

Vaidyanathan S,Watson I D,Jonsson O,Buczynski A Z,Grases F,Heilberg I P,Yasui T,Wyndaele J J,Tozawa K,Kohri K,Schurch B,Hughes P L,Singh G,Soni B M,Sett P,Fraser W D

Publisher

Springer Science and Business Media LLC

Subject

Neurology (clinical),Neurology,General Medicine

Reference19 articles.

1. Ord J, Lunn D, Reynard J . Bladder management and risk of bladder stone formation in spinal cord injured patients. J Urol 2003; 170: 1734–1737.

2. Lamid S, El Ghatit AZ, Melvin JL . Relationship of hypercalciuria to diet and bladder stone formation in spinal cord injury patients. Am J Phys Med 1984; 63: 182–187.

3. Singh PP, Pendse AK, Rathore V, Kiran R, Dashora PK . Interdependence of urinary factors in calcareous bladder stone patients. Int Urol Nephrol 1989; 21: 145–151.

4. Gupta M, Tuncay OL, Valderrama E, Smith AD . Inhibition of calcium oxalate urolithiasis in a rat model of lithogenesis using bisphosphonates. J Endourol 1997; 11: l–4.

5. Bone III HG, Zerwekh JE, Britton F, Pak CY . Treatment of calcium urolithiasis with diphosphonate: efficacy and hazards. J Urol 1979; 121: 568–571.

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