Loss of Trim24 (Tif1α) gene function confers oncogenic activity to retinoic acid receptor alpha-Reference-Cited by-同舟云学术

Loss of Trim24 (Tif1α) gene function confers oncogenic activity to retinoic acid receptor alpha

Published:2007-11-18 Issue:12 Volume:39 Page:1500-1506
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Khetchoumian Konstantin,Teletin Marius,Tisserand Johan,Mark Manuel,Herquel Benjamin,Ignat Mihaela,Zucman-Rossi Jessica,Cammas Florence,Lerouge Thierry,Thibault Christelle,Metzger Daniel,Chambon Pierre,Losson Régine

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng.2007.15.pdf

Reference22 articles.

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2. Mark, M., Ghyselinck, N.B. & Chambon, P. Function of retinoid nuclear receptors: lessons from genetic and pharmacological dissections of the retinoic acid signaling pathway during mouse embryogenesis. Annu. Rev. Pharmacol. Toxicol. 46, 451–480 (2006).

3. Rosenfeld, M.G. & Glass, C.K. Coregulator codes of transcriptional regulation by nuclear receptors. J. Biol. Chem. 276, 36865–36868 (2001).

4. Le Douarin, B., Zechel, C., Garnier, J.-M., Lutz, Y., Tora, L., Pierrat, B., Heery, D., Gronemeyer, H., Chambon, P. & Losson, R. The N-terminal part of TIF1, a putative mediator of the ligand-dependent activation function (AF-2) of nuclear receptors, is fused to B-raf in the oncogenic protein T18. EMBO J. 14, 2020–2033 (1995).

5. Zhong, S. et al. A RA-dependent, tumor-growth suppressive transcription complex is the target of the PML-RARα and T18 oncoproteins. Nat. Genet. 23, 287–295 (1999).

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