NF-κB p50 promotes HIV latency through HDAC recruitment and repression of transcriptional initiation-Reference-Cited by-同舟云学术

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NF-κB p50 promotes HIV latency through HDAC recruitment and repression of transcriptional initiation

Published:2005-12-01 Issue:1 Volume:25 Page:139-149
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Williams Samuel A,Chen Lin-Feng,Kwon Hakju,Ruiz-Jarabo Carmen M,Verdin Eric,Greene Warner C

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference31 articles.

1. The p65 (RelA) Subunit of NF-κB Interacts with the Histone Deacetylase (HDAC) Corepressors HDAC1 and HDAC2 To Negatively Regulate Gene Expression

2. Exchange of N-CoR Corepressor and Tip60 Coactivator Complexes Links Gene Expression by NF-κB and β-Amyloid Precursor Protein

3. NF-κB Binds P-TEFb to Stimulate Transcriptional Elongation by RNA Polymerase II

4. Duration of Nuclear NF-κB Action Regulated by Reversible Acetylation

5. The Human Factors YY1 and LSF Repress the Human Immunodeficiency Virus Type 1 Long Terminal Repeat via Recruitment of Histone Deacetylase 1

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