ATM–Chk2–p53 activation prevents tumorigenesis at an expense of organ homeostasis upon Brca1 deficiency-Reference-Cited by-同舟云学术

ATM–Chk2–p53 activation prevents tumorigenesis at an expense of organ homeostasis upon Brca1 deficiency

Published:2006-05-04 Issue:10 Volume:25 Page:2167-2177
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Cao Liu,Kim Sangsoo,Xiao Cuiying,Wang Rui-Hong,Coumoul Xavier,Wang Xiaoyan,Li Wen Mei,Xu Xiao Ling,De Soto Joseph A,Takai Hiroyuki,Mai Sabine,Elledge Stephen J,Motoyama Noboru,Deng Chu-Xia

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference45 articles.

1. Normal lymphocyte development and thymic lymphoma formation in Brca1 exon-11-deficient mice

2. Atm-Deficient Mice: A Paradigm of Ataxia Telangiectasia

3. Chk1 and Chk2 kinases in checkpoint control and cancer

4. DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis

5. Heterozygous Germ Line hCHK2 Mutations in Li-Fraumeni Syndrome

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