Mxi2 promotes stimulus-independent ERK nuclear translocation-Reference-Cited by-同舟云学术

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Mxi2 promotes stimulus-independent ERK nuclear translocation

Published:2007-01-25 Issue:3 Volume:26 Page:635-646
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Casar Berta,Sanz-Moreno Victoria,Yazicioglu Mustafa N,Rodríguez Javier,Berciano María T,Lafarga Miguel,Cobb Melanie H,Crespo Piero

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference30 articles.

1. Two co-existing mechanisms for nuclear import of MAP kinase: passive diffusion of a monomer and active transport of a dimer

2. Nuclear Export of Map Kinase (ERK) Involves a Map Kinase Kinase (Mek-Dependent) Active Transport Mechanism

3. Subcellular Localization Determines the Protective Effects of Activated ERK2 against Distinct Apoptogenic Stimuli in Myeloid Leukemia Cells

4. A Conserved Docking Site in MEKs Mediates High-affinity Binding to MAP Kinases and Cooperates with a Scaffold Protein to Enhance Signal Transmission

5. Nuclear export of the stress-activated protein kinase p38 mediated by its substrate MAPKAP kinase-2

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