Wound-healing defect of CD18−/− mice due to a decrease in TGF-β1 and myofibroblast differentiation-Reference-Cited by-同舟云学术

Wound-healing defect of CD18−/− mice due to a decrease in TGF-β1 and myofibroblast differentiation

Published:2005-09-08 Issue:19 Volume:24 Page:3400-3410
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Peters Thorsten,Sindrilaru Anca,Hinz Boris,Hinrichs Ralf,Menke André,Al-Azzeh Ezz Al Din,Holzwarth Katrin,Oreshkova Tsvetelina,Wang Honglin,Kess Daniel,Walzog Barbara,Sulyok Silke,Sunderkötter Cord,Friedrich Wilhelm,Wlaschek Meinhard,Krieg Thomas,Scharffetter-Kochanek Karin

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference62 articles.

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3. Trauma-hemorrhage delays wound healing potentially by increasing pro-inflammatory cytokines at the wound site

4. Structure and function of the leukocyte adhesion molecules CD11/CD18

5. Dependence of collagen remodelling on ?-smooth muscle actin expression by fibroblasts

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