Autophosphorylation of ataxia-telangiectasia mutated is regulated by protein phosphatase 2A-Reference-Cited by-同舟云学术

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Autophosphorylation of ataxia-telangiectasia mutated is regulated by protein phosphatase 2A

Published:2004-10-28 Issue:22 Volume:23 Page:4451-4461
ISSN:0261-4189
Container-title:The EMBO Journal
language:
Short-container-title:EMBO J

Author:

Goodarzi Aaron A,Jonnalagadda Jyoti C,Douglas Pauline,Young David,Ye Ruiqiong,Moorhead Greg B G,Lees-Miller Susan P,Khanna Kum Kum

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference39 articles.

1. Requirement of protein phosphatase 5 in DNA-damage-induced ATM activation

2. DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation

3. Phosphatases join kinases in DNA-damage response pathways

4. Enhanced Phosphorylation of p53 by ATM in Response to DNA Damage

5. Nuclear localization of protein phosphatase 5 is dependent on the carboxy-terminal region

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1. TORC2 is required for the accumulation of γH2A in response to DNA damage;Journal of Biological Chemistry;2024-08

2. Key molecular DNA damage responses of human cells to radiation;Frontiers in Cell and Developmental Biology;2024-07-10

3. The ataxia-telangiectasia disease protein ATM controls vesicular protein secretion via CHGA and microtubule dynamics via CRMP5;2024-06-26

4. Potential New Therapies “ROS-Based” in CLL: An Innovative Paradigm in the Induction of Tumor Cell Apoptosis;Antioxidants;2024-04-17

5. The PP2A phosphatase counteracts the function of the 9-1-1 axis in checkpoint activation;Cell Reports;2023-11

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