Thalidomide for rheumatic disease: the best of both worlds?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
http://www.nature.com/articles/ncprheum0504.pdf
Reference12 articles.
1. Dixon WG et al. (2006) Rates of serious infection, including site-specific and bacterial intracellular infection in rheumatoid arthritis patients receiving anti-tumor necrosis factor therapy: results from the British Society for Rheumatology Biologics Register. Arthritis Rheum 54: 2368–76
2. Listing J et al. (2005) Infections in patients with rheumatoid arthritis treated with biologic agents. Arthritis Rheum 52: 3403–3412
3. Stephens TD and Brynner R (2001) Dark Remedy: The impact of thalidomide and its revival as a vital medicine. New York: Perseus Books
4. Majumdar S et al. (2002) Thalidomide suppresses NF-kappa B activation induced by TNF and H2O2, but not that activated by ceramide, lipopolysaccharides, or phorbol ester. J Immunol 168: 2644–2651
5. Sampaio EP et al. (1993) The influence of thalidomide on the clinical and immunologic manifestation of erythema nodosum leprosum. J Infect Dis 168: 408–414
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