Multiple system atrophy
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Medicine
Link
https://www.nature.com/articles/s41572-022-00382-6.pdf
Reference280 articles.
1. Fanciulli, A. & Wenning, G. K. Multiple-system atrophy. N. Engl. J. Med. 372, 249–263 (2015).
2. Quinn, N. Multiple system atrophy–the nature of the beast. J. Neurol. Neurosurg. Psychiatry https://doi.org/10.1136/jnnp.52.suppl.78 (1989).
3. Graham, J. G. & Oppenheimer, D. R. Orthostatic hypotension and nicotine sensitivity in a case of multiple system atrophy. J. Neurol. Neurosurg. Psychiatry 32, 28–34 (1969).
4. Papp, M. I., Kahn, J. E. & Lantos, P. L. Glial cytoplasmic inclusions in the CNS of patients with multiple system atrophy (striatonigral degeneration, olivopontocerebellar atrophy and Shy-Drager syndrome). J. Neurol. Sci. 94, 79–100 (1989). Description of GCIs in patients with striatonigral degeneration, olivopontocerebellar atrophy and Shy–Drager syndrome providing evidence that these three syndromes are different manifestations of MSA.
5. Spillantini, M. G. et al. Filamentous α-synuclein inclusions link multiple system atrophy with Parkinson’s disease and dementia with Lewy bodies. Neurosci. Lett. 251, 205–208 (1998). This is the first report that α-synuclein is the major component of GCI in MSA.
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