The binding capability of plasma phospholipid transfer protein, but not HDL pool size, is critical to repress LPS induced inflammation-Reference-Cited by-同舟云学术

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The binding capability of plasma phospholipid transfer protein, but not HDL pool size, is critical to repress LPS induced inflammation

Published:2016-02-09 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Yu Yang,Cui Yingjie,Zhao Yanan,Liu Shuai,Song Guohua,Jiao Peng,Li Bin,Luo Tian,Guo Shoudong,Zhang Xiangjian,Wang Hao,Jiang Xian-Cheng,Qin Shucun

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep20845.pdf

Reference34 articles.

1. van Deventer, S. J. et al. Experimental endotoxemia in humans: analysis of cytokine release and coagulation, fibrinolytic and complement pathways. Blood 76, 2520–6 (1990).

2. Pirillo, A., Catapano, A. L. & Norata, G. D. HDL in infectious diseases and sepsis. Handb Exp Pharmacol 224, 483–508 (2015).

3. Guo, L. et al. Hepatic scavenger receptor BI protects against polymicrobial-induced sepsis through promoting LPS clearance in mice. J Biol Chem 289, 14666–73 (2014).

4. Levine, D. M. et al. In vivo protection against endotoxin by plasma high density lipoprotein. Proc Natl Acad Sci USA 90, 12040–4 (1993).

5. van Leeuwen, H. J. et al. Lipoprotein metabolism in patients with severe sepsis. Crit Care Med 31, 1359–66 (2003).

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