Over-production of nitric oxide by oxidative stress-induced activation of the TGF-β1/PI3K/Akt pathway in mesangial cells cultured in high glucose-Reference-Cited by-同舟云学术

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Over-production of nitric oxide by oxidative stress-induced activation of the TGF-β1/PI3K/Akt pathway in mesangial cells cultured in high glucose

Published:2013-03-25 Issue:4 Volume:34 Page:507-514
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Zhai Yun-peng,Lu Qian,Liu Yao-wu,Cheng Qian,Wei Ya-qin,Zhang Fan,Li Cheng-lin,Yin Xiao-xing

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

http://www.nature.com/articles/aps2012207.pdf

Reference45 articles.

1. Kanwar YS, Wada J, Sun L, Xie P, Wallner EI, Chen S, et al. Diabetic nephropathy: mechanisms of renal disease progression. Exp Biol Med (Maywood) 2008; 233: 4–11.

2. Ha H, Lee HB . Reactive oxygen species amplify glucose signalling in renal cells cultured under high glucose and in diabetic kidney. Nephrology (Carlton) 2005; Suppl: S7–10.

3. Sedeek M, Callera G, Montezano A, Gutsol A, Heitz F, Szyndralewiez C, et al. Critical role of Nox4-based NADPH oxidase in glucose-induced oxidative stress in the kidney: implications in type 2 diabetic nephropathy. Am J Physiol Renal Physiol 2010; 299: F1348–58.

4. Jaimes EA, Hua P, Tian RX, Raij L . Human glomerular endothelium: interplay among glucose, free fatty acids, angiotensin II, and oxidative stress. Am J Physiol Renal Physiol 2009; 298: F125–32.

5. Brownlee M . The pathobiology of diabetic complications: a unifying mechanism. Diabetes 2005; 54: 1615–25.

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