Imatinib induces H2AX phosphorylation and apoptosis in chronic myelogenous leukemia cells in vitro via caspase-3/Mst1 pathway-Reference-Cited by-同舟云学术

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Imatinib induces H2AX phosphorylation and apoptosis in chronic myelogenous leukemia cells in vitro via caspase-3/Mst1 pathway

Published:2012-03-05 Issue:4 Volume:33 Page:551-557
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Zhang Yan-jun,Lu Cheng-rong,Cao Yan,Luo Yuan,Bao Rong-feng,Yan Shu,Xue Mei,Zhu Feng,Wang Zhe,Duan Lian-ning

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

http://www.nature.com/articles/aps20129.pdf

Reference29 articles.

1. Brauer KM, Werth D, von Schwarzenberg K, Bringmann A, Kanz L, Grunebach F, et al. BCR-ABL activity is critical for the immunogenicity of chronic myelogenous leukemia cells. Cancer Res 2007; 67: 5489–97.

2. Goldman JM, Melo JV . Chronic myeloid leukemia — advances in biology and new approaches to treatment. N Engl J Med 2003; 349: 1451–64.

3. Goldman JM, Melo JV . Targeting the BCR-ABL tyrosine kinase in chronic myeloid leukemia. N Engl J Med 2001; 344: 1084–6.

4. Yoshida C, Yoshida F, Sears DE, Hart SM, Ikebe D, Muto A, et al. Bcr-Abl signaling through the PI-3/S6 kinase pathway inhibits nuclear translocation of the transcription factor Bach2, which represses the antiapoptotic factor heme oxygenase-1. Blood 2007; 109: 1211–9.

5. Strout MP, Schatz DG . Imatinib resistance and progression of CML to blast crisis: somatic hypermutation AIDing the way. Cancer Cell 2009; 16: 174–6.

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