LGI1 acts presynaptically to regulate excitatory synaptic transmission during early postnatal development
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep21769.pdf
Reference27 articles.
1. Kalachikov, S. et al. Mutations in LGI1 cause autosomal-dominant partial epilepsy with auditory features. Nat. Genet. 30, 335–341 (2002).
2. Morante-Redolat, J. M. et al. Mutations in the LGI1/Epitempin gene on 10q24 cause autosomal dominant lateral temporal epilepsy. Hum. Mol. Genet. 11, 1119–1128 (2002).
3. Michelucci, R., Pasini, E. & Nobile, C. Lateral temporal lobe epilepsies: clinical and genetic features. Epilepsia 50 Suppl 5, 52–54 (2009).
4. Irani, S. R. et al. Antibodies to Kv1 potassium channel-complex proteins leucine-rich, glioma inactivated 1 protein and contactin-associated protein-2 in limbic encephalitis, Morvan’s syndrome and acquired neuromyotonia. Brain 133, 2734–2748 (2010).
5. Lai, M. et al. Investigation of LGI1 as the antigen in limbic encephalitis previously attributed to potassium channels: a case series. Lancet. Neurol. 9, 776–785 (2010).
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