Toward dissecting the etiology of schizophrenia: HDAC1 and DAXX regulate GAD67 expression in an in vitro hippocampal GABA neuron model-Reference-Cited by-同舟云学术

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Toward dissecting the etiology of schizophrenia: HDAC1 and DAXX regulate GAD67 expression in an in vitro hippocampal GABA neuron model

Published:2016-01 Issue:1 Volume:6 Page:e723-e723
ISSN:2158-3188
Container-title:Translational Psychiatry
language:en
Short-container-title:Transl Psychiatry

Author:

Subburaju S,Coleman A J,Ruzicka W B,Benes F M

Publisher

Springer Science and Business Media LLC

Subject

Biological Psychiatry,Cellular and Molecular Neuroscience,Psychiatry and Mental health

Link

http://www.nature.com/articles/tp2015224.pdf

Reference50 articles.

1. Murray CJ, Lopez AD . Evidence-based health policy—lessons from the Global Burden of Disease Study. Science 1996; 274: 740–743.

2. Murray RM, Sham P, Van Os J, Zanelli J, Cannon M, McDonald C . A developmental model for similarities and dissimilarities between schizophrenia and bipolar disorder. Schizophr Res 2004; 71: 405–416.

3. Kirov G, O'Donovan MC, Owen MJ . Finding schizophrenia genes. J Clin Invest 2005; 115: 1440–1448.

4. Akbarian S, Kim JJ, Potkin SG, Hagman JO, Tafazzoli A, Bunney WE Jr et al. Gene expression for glutamic acid decarboxylase is reduced without loss of neurons in prefrontal cortex of schizophrenics. Arch Gen Psychiatry 1995; 52: 258–266.

5. Guidotti A, Auta J, Davis JM, Di-Giorgi-Gerevini V, Dwivedi Y, Grayson DR et al. Decrease in reelin and glutamic acid decarboxylase67 (GAD67) expression in schizophrenia and bipolar disorder: a postmortem brain study. Arch Gen Psychiatry 2000; 57: 1061–1069.

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