Genome-wide copy-number variation study of psychosis in Alzheimer’s disease-Reference-Cited by-同舟云学术

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Genome-wide copy-number variation study of psychosis in Alzheimer’s disease

Published:2015-06 Issue:6 Volume:5 Page:e574-e574
ISSN:2158-3188
Container-title:Translational Psychiatry
language:en
Short-container-title:Transl Psychiatry

Author:

Zheng X,Demirci F Y,Barmada M M,Richardson G A,Lopez O L,Sweet R A,Kamboh M I,Feingold E

Publisher

Springer Science and Business Media LLC

Subject

Biological Psychiatry,Cellular and Molecular Neuroscience,Psychiatry and Mental health

Link

http://www.nature.com/articles/tp201564.pdf

Reference66 articles.

1. Gatz M, Reynolds CA, Fratiglioni L, Johansson B, Mortimer JA, Berg S et al. Role of genes and environments for explaining Alzheimer disease. Arch Gen Psychiatry 2006; 63: 168–174.

2. Kamboh MI, Demirci FY, Wang X, Minster RL, Carrasquillo MM, Pankratz VS et al. Genome-wide association study of Alzheimer's disease. Transl Psychiatry 2012; 2: e117.

3. Grupe A, Abraham R, Li Y, Rowland C, Hollingworth P, Morgan A et al. Evidence for novel susceptibility genes for late-onset Alzheimer′s disease from a genome-wide association study of putative functional variants. Hum Mol Genet 2007; 16: 865–873.

4. Coon KD, Myers AJ, Craig DW, Webster JA, Pearson JV, Lince DH et al. A high-density whole-genome association study reveals that APOE is the major susceptibility gene for sporadic late-onset Alzheimer′s disease. J Clin Psychiatry 2007; 68: 613–618.

5. Reiman EM, Webster JA, Myers AJ, Hardy J, Dunckley T, Zismann VL et al. GAB2 alleles modify Alzheimer′s risk in APOE epsilon4 carriers. Neuron 2007; 54: 713–720.

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4. DNA methylation signatures of Alzheimer’s disease neuropathology in the cortex are primarily driven by variation in non-neuronal cell-types;Nature Communications;2022-09-24

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