Biological characteristics comparison of HBV rtA181T mutants with truncated or substituted HBsAg expression in vitro and in vivo model systems
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep39260.pdf
Reference21 articles.
1. Deng, L. & Tang, H. Hepatitis B virus drug resistance to current nucleos(t)ide analogs: Mechanisms and mutation sites. Hepatol Res. 41, 1017–1024 (2011).
2. Liaw, Y. F. Impact of hepatitis B therapy on the long-term outcome of liver disease. Liver Int. 31 Suppl 1, 117–121 (2011).
3. Xu, L. et al. Pre-core/basal-core promoter and reverse transcriptase mutations in chronic HBV infected-patients. Hepatogastroenterology. 59, 212–215 (2012).
4. Villet, S. et al. Impact of hepatitis B virus rtA181V/T mutants on hepatitis B treatment failure. J Hepatol. 48, 747–755 (2008).
5. Huang, Y. & Lok, A. S. Viral factors and outcomes of chronic HBV infection. Am J Gastroenterol. 106, 93–95 (2011).
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2. Detection of the serological markers hepatitis B virus surface antigen (HBsAg) and hepatitis B core IgM antibody (anti‐HBcIgM) in the diagnosis of acute hepatitis B virus infection after recent exposure;Microbiology and Immunology;2021-10-25
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4. Hepatitis B virus rtA181T/sW172non-stop mutation may increase resistance fold to adefovir- and entecavir-resistant mutants compared to rtA181T/sW172* mutation;Antiviral Research;2018-06
5. The truncated mutant HBsAg expression increases the tumorigenesis of hepatitis B virus by regulating TGF-β/Smad signaling pathway;Virology Journal;2018-04-02
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